Secondary to hypertension? -

i am making a nursing diagnoses, can i write there the problem, decreased cardiac output related to increased vascular resistance SECONDARY to hypertension? thnx

I would like to help, but the question is so loosely phrased that it doesn t really make sense. I m not criticizing you, but simply trying to let you know how impossible it is to formulate a guided answer. But here goes: (1) CO has an inverse relationship to TPR. So when CO goes down (typically upon going to sleep for instance, or periods of deep relaxation) then TPR rises. The converse is also true, in that when the body (or mind) is subject to stress -typically after a big heavy meal, or exercising on a treadmill- greater throughput of blood and thus higher CO, is called for, and TPR goes DOWN to facilitate flow, and minimizing the extent to which Mean Arterial Pressure has to rise. (The Mean Pressure Equation, Pm = CO x TPR) (2) Quite what you mean by quot;secondary to hypertensionquot; is unclear. The above mechanism (para.1) MUST go on and continue operate, both when quot;hypertensionquot; is present or in its absence. That is, whether or not the patient is hypertensive doesn t make any difference. The brain and nervous control systems still ensure that TPR increases with decreasing CO and vice versa. So one s not in any way quot;secondaryquot; to the other. They are independent of each other and complementary. which is why acute hypertension can be fatal. A patient can live with severe hypertension, and can carry on a normal relaxed life. Similarly, a person without hypertension can stand severe stresses. But when a hypertensive is subjected to transient excessive stresses, and the heart is CAPABLE of producing (L. ventricular) pressures to satisfy the body s demand for adequate CO under these conditions, THEN blood vessels rupture... Do feel free to make a further posting if you need more clarification.